Every Meal Generates Inflammation That Activates Immune System

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Jan 31, 2017 02:02 PM EST

New research shows that while we are eating food we are not only consuming nutrients but a significant amount of bacteria also entering into our body. In a natural way, our body is simultaneously fighting against these bacterias and circulating the glucose as well. This leads to an inflammatory reaction in the immune system and the body tries to protect this.

Gradually, this inflammatory issue leads to type 2 diabetes in obese people. It also takes the individual to a chronic health disorder with multiple impacts. There conducted many types of research to control the excess production of a chemical which involved in this phenomenon, Interleukin-1beta. The excess production of this substance leads to the chronic inflammation, which results in the death of insulin cells, Times of India reports.

The inflammation does not have many positive benefits, even though it supports to uptake sugar and activate the immune system. The research results revealed in the publication Nature Immunology by the scholars from the Department of Biomedicine at the University Hospital located at Basel, Science Daily reports. They noticed that the "scavenger cells" release messenger chemicals IL-1beta in multiple levels, which depend on the amount of glucose in the blood, India Today.

 It results to accelerate the insulin excretion in pancreatic beta cells. This insulin leads the macrophages or scavenger cells to stimulate the IL-1beta generation. Insulin and IL-1beta are working together to control the quantity of blood sugar. At the same time, the messenger substance in IL-1beta assures that the immune system is receiving sufficient glucose and working actively. "The insulin then causes the macrophages to increase IL-1beta production. Insulin and IL-1beta are unanimously performing to control the quantity of glucose, at the same time messenger chemical IL-1beta ensures that the immune system is supplied with sugar and thus keeps energetic," Dr. Marc Donath at the University Hospital, suggests, Science Daily reports.

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